Clinical manifestations of alcoholic hepatitis

1. symptoms

Alcoholic hepatitis often near future before a concentration and excessive intake of history, there are obvious abdominal distension, body scrub and weakness, loss of appetite, diarrhoea, nausea and vomiting, abdominal pain, weight loss, some patients with fever, leukocytosis (primarily neutrophil number), like bacterial infection. Lischner analysis 169 cases of alcoholic hepatitis, loss of appetite and 77%, 55% of the heart to vomiting, abdominal pain, weight loss 46% 435.

A medical institution admitted 24 cases of alcoholic hepatitis cases, drinking years 8.5 years ~ 21 years. Equivalent to drinking precision 60 ~ 200g/d, average 117g/d. According to the aforementioned pathological grade 24 cases, mild 15 cases of moderate to severe cases, 6 3 cases. Their average duration respectively drinking and the average amount of drinking sperm are obvious differences in the statistics of.

Second, the signs

To jaundice, liver enlargement and tenderness for the characteristic, a few splenomegaly, ascites, pale grey, pink, Spider Nevus, etc. Have liver dysfunction is obvious, ascites occurs neuropsychiatric symptoms. Lischner report 169 cases, hepatomegaly, jaundice 81% and 77%, 59%, fever ascites 56%, 55% of malnutrition, upper gastrointestinal bleeding esophageal varices, 22%, 12%, 10% mental symptoms.

A medical institution admitted 24 cases, abnormal enlargement of 58%, 50% of drinkers face, ganzhang 46%, emaciation, spider 46% 38% 33%, jaundice, splenomegaly, ascites 12.5 21%, 1 case of esophageal varices, esophageal vein show 2 cases, heat 1 cases 38.1 degrees Celsius. Literature reports most hospitalized after as patients with fever, fever also stop within a few days back to normal, but individual patient fever of up to 4 weeks.

As for the pathological changes in light, medium and heavy degree and clinical investigations, the relationship between 24 cases from the laboratory to check the project including AST, ALT, ALP, GGT value increase (mildly alcoholic hepatitis to 43% to 72%; moderate 60%-80%; severe 100% increase), and prothrombin activity decline. Clinical signs: weakness, pain, liver dysfunction, impotence, liver enlargement (mildly alcoholic hepatitis a 36% to 71% hepatomegaly, moderate hepatomegaly 2/3 full with moderate or severe hepatomegaly) are also available for reference.

These checks are non-specific, so specific patients, only rely on these non-specific changes are to distinguish light, moderate and severe is not exact, conditional should do a liver biopsy to help distinguish the severity level.

(Editors: Han 1m1m SWE)